Apparatus and method for treating atrial fibrillation and atrial tachycardia

• US 7,983,748 B2
• Filed: 02/26/2009
• Issued: 07/19/2011
• Est. Priority Date: 02/26/2008
• Status: Expired due to Fees

• Current Assignee: HAK Consulting, LLC
• Original Assignee: HAK Consulting, LLC
• Inventors: Ruse, Richard B.
• Primary Examiner(s): Manuel; George
• Assistant Examiner(s): Wieland; Robert N
• Application Number:
  US12/393,899
• Publication Number:
  US 20090216290A1
• Time in Patent Office: 873 Days
• Field of Search: 607/2, 607/14, 607/44, 607/45, 607/9, 600/509
• US Class Current: 607/2
• CPC Class Codes: A61N 1/20 continuous direct currents
• CPC Class Codes: A61N 1/205 for promoting a biological process
• CPC Class Codes: A61N 1/36114 Cardiac control, e.g. by vagal stimulation stimulating the heart A61N1/362
• CPC Class Codes: A61N 1/3624 occurring in the atrium, i.e. atrial tachycardia
• CPC Class Codes: A61N 1/37223 Circuits for electromagnetic coupling

Abstract

A method and device for treating an electrical problem in an organ, especially a heart, of a human or animal patient comprises atraumatically blocking the transmission of one or more electrical signals external to the organ. Nerve cell membranes near a cathode are depolarized while nerve cell membranes near an anode are hyperpolarized, inducing a DC conduction block. The method and device are especially suitable for treating atrial tachycardia where unwanted signals from at least one pulmonary vein and/or at least one fat pad are blocked within the construct of the heart.

Claims

1. A method for treating an electrical problem in a human or animal patient'"'"'s heart having a left atrium which comprises atraumatically blocking the transmission of one or more electrical signals from a pulmonary vein, a fat pad, or a pulmonary vein and a fat pad from reaching the left atrium of the heart, wherein the one or more electrical signals are blocked by a DC current, voltage, or both DC current and voltage transmitted from an optical or light energy source to at least one noise cancellation device positioned on or adjacent to a pulmonary vein or a fat pad connected to the left atrium.

2. The method of claim 1, wherein nerve cell membranes near a cathode are depolarized while nerve cell membranes near an anode are hyperpolarized, inducing a DC conduction block.

3. The method of claim 1, wherein the DC current, voltage, or DC current and voltage results from a neurostimulator.

4. The method of claim 1, wherein the electrical signals are cancelled, redirected, or both.

5. A system for carrying out the method of claim 1, comprising:

a source of light or optical energy,

a transmitter for transmitting said energy; and

a receiver capable of receiving said energy and using said energy to block electrical signals from a pulmonary vein, a fat pad, or a pulmonary vein and a fat pad from reaching the left atrium of the heart,

wherein the one or more electrical signals are blocked by a DC current, voltage, or both DC current and voltage transmitted from an optical or light energy source.

6. The device of claim 5, wherein the electrical signals are cancelled, directed away from the heart, or a combination thereof.

7. The method of claim 1, wherein the electrical problem is atrial tachycardia, atrial fibrillation, or atrial flutter.

8. The method of claim 7, wherein the atrial flutter is typical atrial flutter or atypical atrial flutter.

9. The method of claim 1, wherein the one or more electrical signals are blocked so that they do not interfere with electrical signals from an SA node and/or the atrial electrical conduction system.

10. A system for treating a cardiac condition in a patient, which comprises:

an ECG sensing and control circuit;

a transmitter for transmitting an optical or light energy signal; and

at least one receiver for receiving said signal,

wherein each receiver is positioned on or adjacent to a pulmonary vein or fat pad and each receiver acts to cancel unwanted electrical signals from the pulmonary vein or fat pad to block the signals from reaching the left atrium of the patient'"'"'s heart.

11. The system of claim 10, wherein the receiver is a neurostimulator.

12. The system of claim 10, wherein nerve cells near a cathode band or electrode are depolarized and nerve cells near an anode band or electrode are hyperpolarized.

13. The system of claim 12, wherein the nerves are within, on, or within and on the surface of pulmonary veins, between two electrical contacts or points.

14. The system of claim 10, which comprises a flexible circuit which is hard wired between the ECG sensing and control circuit and the targeted area for noise cancellation on the heart epicardially.

15. The system of claim 14, wherein the hard wired ECG and control circuits circuit are a neurostimulator.

16. A system for treating a cardiac condition in a patient, which comprises a magnetic neurostimulator comprising two fixed magnets, a traversing magnet, and a wire coil, wherein the traversing magnet oscillates back and forth through the wire coil thereby inducing an alternating current which is rectified through semiconductor diodes to form a direct current electrical voltage and current which comprises a conduction blocking signal wherein each magnetic device is positioned on a pulmonary vein and each magnetic neurostimulator acts to cancel unwanted electrical signals from a pulmonary vein or fat pad to block the signals from reaching the left atrium of the patient'"'"'s heart.

17. The system of claim 16, wherein the traversing magnet DC power supply delivers a DC conduction block as a self-contained power supply or power source for delivery of the DC conduction block.

18. The system of claim 17, wherein the DC conduction block voltage and current increase and decrease as the heart rate increases and decreases in a human heart within medically therapeutic ranges to convert or prevent atrial fibrillation or atrial tachycardia.

19. The system of claim 18, wherein the traversing magnetic power supply provides increased prevention and protection from atrial fibrillation or atrial tachycardia via the heart rate increase causing an increasing neurostimulator output voltage and current up to a predetermined and clamped level while also providing prevention and conversion for atrial fibrillation or atrial tachycardia when the heart is at rest or beating at slower rates.